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Thursday, July 30, 2020 | History

4 edition of The effects of a short period of hypoxia on the ventilatory response to carbon dioxide found in the catalog.

The effects of a short period of hypoxia on the ventilatory response to carbon dioxide

Eryn Patrice Corriveau

The effects of a short period of hypoxia on the ventilatory response to carbon dioxide

by Eryn Patrice Corriveau

  • 243 Want to read
  • 11 Currently reading

Published by National Library of Canada in Ottawa .
Written in English


Edition Notes

Thesis (M.Sc.) -- University of Toronto, 1998.

SeriesCanadian theses = -- Thèses canadiennes
The Physical Object
FormatMicroform
Pagination2 microfiches : negative. --
ID Numbers
Open LibraryOL19347761M
ISBN 100612340864
OCLC/WorldCa46581028

  This article gives an overview of the respiratory failures hypoxemia, hypercapnia and hypoxia. It is essential to understand the various reflex mechanisms & manage any impairment in them. Partial pressure of gases, alveolar-arterial gradient, tissue hypoxia, hypercapnia. Read more here! A short episode of hypoxia (duration.

  Hemolymph gas parameters were evaluated using a point-of-care analyzer in healthy American horseshoe crabs (Limulus polyphemus) at rest and after short- and long-term removal from ne vascular pH, partial pressure of oxygen (PO 2), partial pressure of carbon dioxide, bicarbonate, base excess, total carbon dioxide, and lactate concentrations were determined from . Respiratory effects. Isoflurane causes respiratory depression and diminishes the ventilatory response to carbon dioxide. Although it irritates the upper airway, it is a bronchodilator. Cardiovascular effects. Anaesthetic concentrations of isoflurane, i.e. 1– MAC, cause only slight impairment of myocardial contractility. Isoflurane causes.

EDITOR—Dubowitz's study of the effect of temazepam on oxygen saturation at high altitude found that benzodiazepines do not have a depressant effect.1 He explains the discrepancy between his findings and those of previous studies by the fact that other studies have investigated the effect of long acting benzodiazepines.2 Dubowitz's probands were investigated after altitude acclimatisation. Carbon dioxide is a byproduct of the energy-generating chemical reactions occurring within the cells of the body. Carbon dioxide produced by these chemical reactions within the cells of the body diffuses into blood which transports it into the lungs where it is eliminated from the body in exhaled air.


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The effects of a short period of hypoxia on the ventilatory response to carbon dioxide by Eryn Patrice Corriveau Download PDF EPUB FB2

The respiratory response of the new-born lamb to inhaled CO-2 with and without accompanying hypoxia. J Physiol. Jul; (1)– [PMC free article] Rigatto H, Brady JP, de la Torre Verduzco R. Chemoreceptor reflexes in preterm infants: II.

The effect of gestational and postnatal age on the ventilatory response to inhaled carbon by:   While many studies in the past have characterised the effect of hypoxia on the ventilatory response to carbon dioxide in terms of the sensitivities of the chemoreflexes, the effect of hypoxia on chemoreflex carbon dioxide thresholds themselves and on ventilation at partial pressures below these thresholds is less well studied and the separation of central and peripheral chemoreflex drives to Cited by: THE EFFECTS OF A SHORT PERIOD OF EYPOXIA ON THE VENTILATORY RESPONSE TO CARBON DIOXIDE.

Master of Science Eryn Patrice Corn-veau Department of Physiology, University of Toronto In the present study, a modified rebreathing technique was used to characterize the ventilatory response to carbon dioxide before and after exposure to either 20 minutes of room air or Author: Eryn Patrice Corriveau.

Initially, ventilation increases during hypoxia, but after several minutes, ventilation decreases and apnea may ensue. 5 The ventilatory response to carbon dioxide is decreased in the micropremie, and hypoxia further blunts this response. 6 Anesthetic drugs depress the ventilatory responses to both hypoxia and hypercapnia.

Hypoxia and hypercapnia occur commonly as a result of apnea and. The optimal IH regime for increasing the ventilatory response in humans is unknown, although in animals there is evidence that multiple short duration bouts of intermittent hypoxia (SDIH) provoke larger changes in chemosensitivity than longer duration bouts of intermittent hypoxia (LDIH).Cited by: 4.

Acute hypoxic ventilatory response. The acute response to hypoxia is an immediate augmentation of ventilatory activity following the onset of a hypoxic stimulus, and typically includes increases in both f R and V T relative to baseline ().To date, almost all animals studied exhibit such a hyperbolic increase in ventilation as a function of decreasing arterial oxygen saturation (Pa O2) (30, but.

Respiratory physiologists traditionally attribute the increased ventilatory response to hypoxia to increased discharge by the carotid-body chemoreceptor, which is. Dona F. Boggs's 36 research works with citations and 1, reads, including: Ventilatory and metabolic responses of burrowing owls, Athene cunicularia, to moderate and extreme hypoxia.

This ventilatory response to hypoxia (HVR) in mammals is the subject of this review. The period immediately after birth contains a critical time window in which environmental factors can cause long-term changes in the structural and functional properties of the respiratory system, resulting in.

At sea level, subjects first breathed room air that was followed by minute steady-state poikilocapnic hypoxia (FIO2 = ). Ventilatory responses to acute poikilocapnic hypoxia (APH) were collected over the first minutes, and responses to chronic poikilocapnic hypoxia (CPH) were collected over the last 3 minutes of the hypoxia exposure.

In the current study, we investigated the effects of intravenous morphine on the steady-state ventilatory responses to step-wise changes in end-tidal oxygen concentration (P ET O 2) against the background of isocapnia in anesthetized also determined the ventilatory response to carbon dioxide and its central and peripheral components using the dynamic end-tidal forcing (DEF) technique).

The ventilatory response to breathing 3 per cent and 5 per cent carbon dioxide in either a hypoxic gas mixture (oxygen = 10 per cent) or a hyperoxic gas mixture (oxygen > 2 per cent) was measured in normal individuals (group I) and in 3 groups of patients with chronic obstructive lung disease: those who demonstrated hypoxia but no previous episodes of carbon dioxide retention (group II), those.

In normoxic conditions, a reduction in arterial carbon dioxide tension causes cerebral vasoconstriction, thereby reducing cerebral blood flow and modifying dynamic cerebral autoregulation (dCA).

It is unclear to what extent these effects are altered by acute hypoxia and the associated hypoxic ventilatory response (respiratory chemoreflex). Since opioid receptors are abundant in brainstem respiratory centers, various opioid-related effects are observed: depression of the ventilatory responses to hypercapnia and hypoxia, depression of ventilation (related to reductions in respiratory rate and volume) and the wakefulness drive to breathe, irregular breathing, suppression of pharyngeal muscle (including the tongue) function.

The acute hypoxic ventilatory response is extremely rapid and ventilation increases within a single breath. However, if hypoxia persists, the response changes in magnitude and pattern (frequency vs.

tidal volume), revealing several time domains of the hypoxic ventilatory response (Figure 6).For example, short-term potentiation of ventilation increases tidal volume while short-term depression.

While the response to the step into hypoxia shows a pronounced overshoot, in comparsion, the response to a step out of hypoxia shows less of an undershoot.

We have used curve fitting techniques to fit a mathematical model to ventilatory response data in order to quantify the differences in the response.

the hypoxic ventilatory response (HVR) is characterized by an abrupt increase in minute ventilation during exposure to hypoxia for a short duration (e.g., 5 min) (33, 44). Hypoxia is a condition in which the body or a region of the body is deprived of adequate oxygen supply at the tissue level.

Hypoxia may be classified as either generalized, affecting the whole body, or local, affecting a region of the body. Although hypoxia is often a pathological condition, variations in arterial oxygen concentrations can be part of the normal physiology, for example, during.

A group of five mice was trained to press a lever to remove 4% CO2 from its inspired air. The effect of six levels of hypoxia upon the latent period for learned escape from the added CO2 was determined.

Mild hypoxia produced a shortened latent period, severe hypoxia a lengthened latent period. The correlation between the behavioral and ventilatory responses to CO2 and hypoxia are. The ventilatory response to carbon dioxide increases with both postnatal and gestational age (see Chapter 3, Respiratory Physiology in Infants and Children) (Rigatto et al., ).

Although hypoxia may inflict long-term consequences on the fetus and newborn, hyperoxia can also cause significant morbidity, especially for preterm infants. Exp Physiol pp – Experimental Physiology Effects of enhanced human chemosensitivity on ventilatory responses to exercise Glen E.

Foster 1,Donald C. McKenzie,2 and A. William Sheel1 1School of Human Kinetics and 2Faculty of Medicine, The University of British Columbia, Vancouver, British Columbia, Canada It is not clear what the effects of different types of intermittent.Ventilatory response to sustained hypoxia A continued presence in a hypoxic environment of more than 24 hours leads to a steady flow of ventilation.

[11] This contingency in the environment causes hypocapnia which decreases ventilation.Inert gas asphyxiation is a form of asphyxiation which results from breathing a physiologically inert gas in the absence of oxygen, or a low amount of oxygen, rather than atmospheric air (which is largely composed of nitrogen and oxygen).

Examples of physiologically inert gases, which have caused accidental or deliberate death by this mechanism, are argon, helium, nitrogen and methane.